Abstract
Chronic neuropathic pain (CNP) causes disability worldwide, but its molecular mechanisms are poorly understood. Despite its prevalence, the molecular basis of CNP remains unclear, hindering diagnosis and treatment. This thesis explores mechanisms underlying CNP cell signalling, treatments, and phenotype.| Date of Award | 17 Jul 2026 |
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| Original language | English |
| Supervisor | Patrick McHugh (Main Supervisor) & James Boyne (Co-Supervisor) |